[1] Saltiel, A. R., & Olefsky, J. M. (2017). Inflammatory mechanisms linking obesity and metabolic disease.
Journal of Clinical Investigation, 127(1), 1–4.
https://doi.org/10.1172/JCI92035
[2] Lindström, J., Absetz, P., Hemio, K., Peltomäki, P., & Peltonen, M. (2010). Reducing the risk of type 2 diabetes with nutrition and physical activity: Efficacy and implementation of lifestyle interventions in Finland.
Public Health Nutrition, 13(6), 993–999.
https://doi.org/10.1017/S1368980010000960
[3] Wasserman, D. H., & Ayala, J. E. (2005). Interaction of physiological mechanisms in control of muscle glucose uptake.
Clinical and Experimental Pharmacology and Physiology, 32(4), 319–323.
https://doi.org/10.1111/j.1440-1681.2005.04191.x
[4] Seldin, M. M., Tan, S. Y., & Wong, G. W. (2014). Metabolic function of the CTRP family of hormones.
Reviews in Endocrine and Metabolic Disorders, 15(2), 111–123.
https://doi.org/10.1007/s11154-013-9255-7
[5] Shapiro, L., & Scherer, P. E. (1998). The crystal structure of a complement-1q family protein suggests an evolutionary link to tumor necrosis factor.
Current Biology, 8(6), 335–338.
https://doi.org/10.1016/S0960-9822(98)70133-2
[6] Park, S. Y., Choi, J. H., Ryu, H. S., Pak, Y. K., Park, K. S., Lee, H. K., & Lee, W. (2009). C1q tumor necrosis factor-related protein isoform 5 is increased in mitochondrial DNA-depleted myocytes and activates AMP-activated protein kinase.
Journal of Biological Chemistry, 284(41), 27780–27789.
https://doi.org/10.1074/jbc.M109.005611
[7] Moller, D. E., & Kaufman, K. D. (2005). Metabolic syndrome: A clinical and molecular perspective.
Annual Review of Medicine, 56, 45–62.
https://doi.org/10.1146/annurev.med.56.082103.104751
[8] Lim, S., Choi, S. H., Koo, B. K., Kang, S. M., Yoon, J. W., Jang, H. C., Choi, S. M., Lee, M. G., Lee, W., Shin, H., Kim, Y. B., Lee, H. K., & Park, K. S. (2012). Effects of aerobic exercise training on C1q tumor necrosis factor α-related protein isoform 5 (myonectin): Association with insulin resistance and mitochondrial DNA density in women.
Journal of Clinical Endocrinology & Metabolism, 97(1), E88–E93.
https://doi.org/10.1210/jc.2011-1743
[9] Choe, H. Y., Park, J. W., Lee, N., Hwang, S. Y., Cho, G. J., Hong, H. C., Yoo, H. J., Hwang, T. G., Kim, S. M., Baik, S. H., Park, K. S., Youn, B. S., & Choi, K. M. (2013). Effects of a combined aerobic and resistance exercise program on C1q/TNF-related protein-3 (CTRP-3) and CTRP-5.
Diabetes Care, 36(10), 3321–3327.
https://doi.org/10.2337/dc13-0178
[10] Gibala, M. J., Little, J. P., Macdonald, M. J., & Hawley, J. A. (2012). Physiological adaptations to low-volume high-intensity interval training in health and disease.
Journal of Physiology, 590(5), 1077–1084.
https://doi.org/10.1113/jphysiol.2011.224725
[11] Heydari, M., Freund, J., & Boutcher, S. H. (2012). The effect of high-intensity intermittent exercise on body composition of overweight young males.
Journal of Obesity, 2012, 1–8.
https://doi.org/10.1155/2012/480467
[12] Glaister, M., Hauck, H., Abraham, C. S., Merry, K. L., Beaver, D., Woods, B., & McInnes, G. (2009). Familiarization, reliability, and comparability of a 40-m maximal shuttle run test. Journal of Sports Science and Medicine, 9(1), 77–82.
[13] Kraniou, G. N., Cameron-Smith, D., & Hargreaves, M. (2006). Acute exercise and GLUT4 expression in human skeletal muscle: Influence of exercise intensity.
Journal of Applied Physiology, 101(3), 934–937.
https://doi.org/10.1152/japplphysiol.01489.2005
[14] Schäffler, A., & Buechler, C. (2012). CTRP family: Linking immunity to metabolism.
Trends in Endocrinology and Metabolism, 23(4), 194–204.
https://doi.org/10.1016/j.tem.2011.12.003
[15] Haghshenas, R., Ravasi, A. A., Kordi, M. R., Hedayati, M., Shabkhiz, F., & Shariatzadeh, M. (2013). The effect of a 12-week endurance training on IL-6, IL-10, and Nesfatin-1 plasma levels of obese male rats. Sport Sciences, 5(4), 109–122.
[16] Goodwin, M. L. (2010). Blood glucose regulation during prolonged, submaximal, continuous exercise: A guide for clinicians.
Journal of Diabetes Science and Technology, 4(3), 694–705.
https://doi.org/10.1177/193229681000400325